P2875
Eur Respir J 1993 Apr;6(4):547-51
Respiration after phrenicotomy and hydrocortisone treatment in
anaesthetized rats.
Nachazel J, Palecek F.
Institute of Pathophysiology, 2nd Medical Faculty, Charles University,
Praha, Czechoslovakia.
The study was designed to determine the extent to which respiratory
muscle wasting, resulting from corticosteroid-induced atrophy, may
affect respiration in normal rats and in rats with denervated diaphragm.
Twenty four male Wistar rats were divided into four groups: 1) controls
with sham operation (SX) and vehicle injections: 2) SX with eight
hydrocortisone (HC) injections (60 mg.kg-1.day-1 i.m.); 3)
phrenicotomized (PX), injected with vehicle; 4) PX and HC-treated. HC
treatment was started on the thirteenth day after surgery. Under
urethane anaesthesia, tidal volume, respiratory rate, arterial carbon
dioxide tension (PaCO2) and occlusion pressure were measured at rest and
after 5 min of stimulated-breathing induced by added dead space 22nd day
after surgery. All HC-treated animals decreased body weight by 32%
compared to untreated rats. The diaphragm weight was reduced in PX rats
by 29%, and after HC by 44%, while in PX rats with HC treatment
diaphragm weight decreased by only 21%. PX rats (HC-untreated) had the
lowest minute ventilation and occlusion pressure. There was no
difference in ventilation between control and both HC-treated groups at
rest. However, ventilation in PX and HC-treated rats did not increase
upon stimulation, and the occlusion pressure increased significantly
only in the HC-untreated animals. We conclude that in the rat, HC
treatment did not affect resting ventilation, but it impaired
ventilation performance, during increased demand, in animals handicapped
by diaphragm denervation.
Comment in:
Eur Respir J. 1993 Apr;6(4):465-6.